[quote="Lizzy"]
Ja, ik geloof hier dus geen hout van, van dat drinkwater, wat ze op wikipedia schrijven. Want dat zou dan namelijk totaal niet verklaren waarom Leo veel gaatjes heeft en geen tandsteen (een zuur speeksel) en ik geen gaatjes heb en wel veel tandsteen. Idem dito qua honden. En wij drinken toch allemaal echt hetzelfde drinkwater!
[/quote]
Er staat toch niet dat daarmee alles verklaard is? Er staat alleen dat het een factor in het geheel is. Zuurgraad in de mond is een belangrijker factor, maar ook niet de enige.
En idd kan iedereen van alles schrijven op Wikipedia. En ik wil niet lullig doen, maar ook jij gebruikt hier Wikipedia wel eens om een punt te maken. Dus .... 8)
Dan maar een universiteits-site:
http://www.ncl.ac.uk/dental/oralbiol/or ... lculus.htm
"What is dental calculus?
Calculus is a deposit of calcium phosphate salts on the surface of teeth. ...
There is no fixed composition of dental calculus because the type and amount of calcium phosphate salt which precipitates onto the tooth surface is affected by a variety of local factors such as
1. the concentration of calcium and phosphate
2. the relative amounts of each ion present locally
3. the pH
4. the presence of other ionic species such as magnesium
5. the presence of other calcium phosphate mineral
I could go on, in fact almost any local condition you care to name will have at least some effect on the type of mineral which deposits. It is, therefore, difficult, or even impossible, to be definitive about calculus composition beyond the fact that it comprises a mixture of calcium phosphate minerals such as:
1. brushite
2. octacalcium phosphate
3. tricalcium phosphate
4. biological apatite
Calculus Formation
Calculus forms wherever a solution of calcium and phosphate becomes unstable. ...
Calculus forms most readily within dental plaque in fact it is often referred to as mineralised plaque. The surface of calculus is rough and provides an excellent site for further plaque growth which in turn may become mineralised. Calculus then often has a layered structure.
Dental plaque
Dental plaque is a firmly adherent microbial biofilm attached to teeth. It is the prime aetiological agent of dental caries and periodontal disease. Careful observation of the distribution of plaque in the mouth reveals that it forms most readily only at certain sites, viz, molar fissures, the gingival crevice and on those surfaces between adjacent teeth (approximally).
Plaque initiation
Sticking to a surface has very real benefits for many microorganisms, they maintain their position in the environment, they can exploit the organic and inorganic molecules which adsorb to surfaces and they can interact with other bacteria in cooperative consortia to maximise their potential.
Plaque formation
Although dental plaque varies considerably in composition, it has been possible to piece together a sequence of events which lead to its establishment. The consensus view of plaque development begins with a clean tooth surface covered by a conditioning film of salivary proteins and glycoproteins, called the tooth pellicle, being colonised by so-called "pioneer species". These multiply forming, first a monolayer and, subsequently, palisades of cells perpendicular to the tooth surface.
During and after this outgrowth period secondary colonisation by a variety of Gram positive and negative species occurs leading to a large increase in the species diversity. Foremost among the events contributing to this secondary colonisation is the process known as co-aggregation whereby colonising microbes attach to cells already part of the developing biofilm. This allows species which can not attach, or can attach only poorly, to the tooth pellicle to participate in the biofilm. At 24 hours the maturing dental plaque contains a wide variety of bacteria and it is possible to detect easily identifiable inter-species associations such as the well documented "corn-cob-configurations", although a wide variety of other inter-species associations will be present.
Further colonisation and growth of established bacteria takes place as the plaque matures to form a stable, climax, community. This pattern of development leading to a climax community has been termed "bacterial succession". The resulting community consists of individual microbes and microcolonies acting in complex consortia which can convey a range of beneficial properties. These include feeding synergies, improved antibiotic resistance and a host of cooperative mechanisms which are the subject of much current research.
High protein diet
Plaque formed on the teeth of individuals with a low carbohydrate, high protein diet, contains fewer acidogenic-aciduric organisms. The pH gradient will be different and the overall pH of the plaque alkaline because of the ammonia produced as a by-product of amino acid breakdown. The higher pH of the plaque will itself inhibit acidogenesis and favour Gram negative organisms which will be present in greater numbers. The proteolytic nature of the plaque will result in the presence of particular peptides such as putrescene and cadaverine which have a characteristic offensive odour."